Dengue Shock Syndrome manifestation of the child consists of
(1) pale skin, cold and humid, especially on the tip of the toes, hands and nose, while a blue nails. This is caused by the circulation insufisien activity which caused the rise in simpatikus reflex,
(2) children that fussy, fractious and awareness decreased slowly become apatic, sopor and coma. This is due to circulation failure in cerebral;
(3) changes in pulse, both the frequency of any amplitudo. Pulse quickly become soft and can not be touched up because circulation collapts;
(4) tension decreased to 20 mm Hg or less;
(5) the child sistolik pressure be decreased 80 mm Hg or less, and
(6) because of oliguria to anuria perfusi decreasing arterial renalis blood .
During clinical shock, hematocryt is increasing until 20% or greater than the value hematocryt healing. Trombositopenia (less than 10.000/mm3) leukositosi, long bleeding time, and decreased protrombin rate also found during shock. Fibrinogen level may be subnormal and products fibrin fraction increased .
Aberration is being increased serum transaminase level, complement consumption, asidosis metabolik light with hiponatremia, and sometimes hipokoremia, slightly increased serum urea nitrogen, and hipoalbunemia. Roentgenogram chest shows effusion of pleura in almost all patients .
At approximately one third of patients with fever after DHF take several days, the common patients suddenly worsen. This usually happens at or after the fever decreases, that is between the day-to-3-7 . Found on the sign of circulatory failure, touched up skin moist and cold, sianosis around the mouth, rapid pulse and become soft and finally a decrease in blood pressure. People look haggard, nervous, and quickly enter into a critical phase of shock. People often complain pain in the stomach shortly before shock arise.
(1) pale skin, cold and humid, especially on the tip of the toes, hands and nose, while a blue nails. This is caused by the circulation insufisien activity which caused the rise in simpatikus reflex,
(2) children that fussy, fractious and awareness decreased slowly become apatic, sopor and coma. This is due to circulation failure in cerebral;
(3) changes in pulse, both the frequency of any amplitudo. Pulse quickly become soft and can not be touched up because circulation collapts;
(4) tension decreased to 20 mm Hg or less;
(5) the child sistolik pressure be decreased 80 mm Hg or less, and
(6) because of oliguria to anuria perfusi decreasing arterial renalis blood .
During clinical shock, hematocryt is increasing until 20% or greater than the value hematocryt healing. Trombositopenia (less than 10.000/mm3) leukositosi, long bleeding time, and decreased protrombin rate also found during shock. Fibrinogen level may be subnormal and products fibrin fraction increased .
Aberration is being increased serum transaminase level, complement consumption, asidosis metabolik light with hiponatremia, and sometimes hipokoremia, slightly increased serum urea nitrogen, and hipoalbunemia. Roentgenogram chest shows effusion of pleura in almost all patients .
At approximately one third of patients with fever after DHF take several days, the common patients suddenly worsen. This usually happens at or after the fever decreases, that is between the day-to-3-7 . Found on the sign of circulatory failure, touched up skin moist and cold, sianosis around the mouth, rapid pulse and become soft and finally a decrease in blood pressure. People look haggard, nervous, and quickly enter into a critical phase of shock. People often complain pain in the stomach shortly before shock arise.
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